Scott h reiniger biography of abraham lincoln

  • Scott H. Reiniger, born in New York City, holds a Bachelor of Arts Degree in Theater Arts majoring in Acting/Directing at Rollins College in Florida.
  • This paper unveils the revolutionary potential incarnated in the post-9/11 transformed figure of the cinematic zombie.
  • PLCγ2 dysfunction is associated with a variety of diseases including cancer, neurodegeneration, and immune disorders.
  • On Violence

    Abstract

    This paper unveils the revolutionary potential incarnated in the post-9/11 transformed figure of the cinematic zombie. It is my contention that zombies, through their cinematic (r)evolution, came to embody Deleuze and Guattari’s vision of the nomad war machine. Zombie films are used as a vehicle for addressing the tension between the hegemonic fear of the violent multitude in revolt and the counter-hegemonic liberatory potential of the rising masses. It is impossible to achieve a final resolution between these contradicting tendencies since the narrative structure of zombie films remains open-ended. The characteristics of the zombies and the meaning ascribed to them transform over time but they also maintain a continuity with a difference with the previous expressions of the monstrous. The monstrous characteristics which have pertained since George A. Romero’s paradigm shift in the 1960s (the zombifying contagion, violence and swarm attacks), joined with the new features appearing in the American zombie cinema of the new millennium, formulate a response to the manifest and latent violence of the State apparatus.

    Keywords: zombies, film, hegemony, nomad war machine, multitude, State apparatus

    Introduction

    The figure of a zombie continues to invade al

  • scott h reiniger biography of abraham lincoln
  • Abstract

    Phosphatidylinositol-specific phospholipase Cγ2 (PLCγ2) is a critical signaling molecule activated downstream from a variety of cell surface receptors that contain an intracellular immunoreceptor tyrosine-based activation motif. These receptors recruit kinases such as Syk, BTK, and BLNK to phosphorylate and activate PLCγ2, which then generates 1D-myo-inositol 1,4,5-trisphosphate and diacylglycerol. These well-known second messengers are required for diverse membrane functionality including cellular proliferation, endocytosis, and calcium flux. As a result, PLCγ2 dysfunction is associated with a variety of diseases including cancer, neurodegeneration, and immune disorders. The diverse pathologies associated with PLCγ2 are exemplified by distinct genetic variants. Inherited mutations at this locus cause PLCγ2-associated antibody deficiency and immune dysregulation, in some cases with autoinflammation. Acquired mutations at this locus, which often arise as a result of BTK inhibition to treat chronic lymphocytic leukemia, result in constitutive downstream signaling and lymphocyte proliferation. Finally, a third group of PLCγ2 variants actually has a protective effect in a variety of neurodegenerative disorders, presumably by increased uptake and degradation of deleteriou

    The 200 Principal Horror Movies of Lessening Time

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