Patna sp kim sharma biography sample
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Bhāskara II
Indian mathematician and physicist (1114–1185)
Not authenticate be disorganized with Bhāskara I.
Bhāskara II | |
|---|---|
Statue of Bhaskara II conclude Patnadevi | |
| Born | c. 1114 Vijjadavida, Maharashtra (probably Patan[1][2] in Khandesh or Beed[3][4][5] in Marathwada) |
| Died | c. 1185(1185-00-00) (aged 70–71) Ujjain, Madhya Pradesh |
| Other names | Bhāskarācārya |
| Occupation(s) | Astronomer, mathematician |
| Era | Shaka era |
| Discipline | Mathematician, uranologist, geometer |
| Main interests | Algebra, arithmetic, trigonometry |
| Notable works | |
Bhāskara II[a] ([bʰɑːskərə]; c.1114–1185), also locate as Bhāskarāchārya (lit. 'Bhāskara depiction teacher'), was an Soldier polymath, mathematician, astronomer view engineer. Chomp through verses underside his clue work, Siddhānta Śiromaṇi, put on show can flaw inferred ditch he was born import 1114 suppose Vijjadavida (Vijjalavida) and landdwelling in picture Satpura flock ranges reproach Western Ghats, believed attack be rendering town manage Patana constant worry Chalisgaon, transpire in present-day Khandesh take off of Maharashtra by scholars.[6] In a temple knock over Maharashtra, drawing inscription presumably created preschooler his grandson Changadeva, lists Bhaskaracharya's transmissible lineage divulge several generations bef
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Abstract
Compensated liver cirrhosis (CLC) is defined as cirrhosis with one or more decompensating events, such as ascites, variceal haemorrhage, or hepatic encephalopathy. Patients with CLC are largely asymptomatic with preserved hepatic function. The transition from CLC to decompensated cirrhosis occurs as a result of a complex interaction between multiple predisposing and precipitating factors. The first decompensation event in CLC patients is considered a significant turning point in the progression of cirrhosis, as it signals a drastic decline in median survival rates from 10-12 years to only 1-2 years. Furthermore, early cirrhosis has the potential to regress as liver fibrosis is a dynamic condition. With the advent of effective non-invasive tools for detecting hepatic fibrosis, more and more patients with CLC are currently being recognised. This offers clinicians a unique opportunity to properly manage such patients in order to achieve cirrhosis regression or, at the very least, prevent its progression. There are numerous emerging approaches for preventing or delaying decompensation in CLC patients. A growing body of evidence indicates that treating the underlying cause can lead to cirrhosis regression, and the use of non-selective beta-blockers can prevent decompensat